第一篇：申請國外博士 個人簡歷(CV)

XXX

Gender:Male

Date of Birth: 10/20/1987Add: Nanjing University of Information Science & Technology

School of Environmental Science and EngineeringSchool Office

No.219, Ningliu Road, Pukou District, Nanjing, Jiangsu Province, 210044, P.R.ChinaE-mail:

Tel: 86-***EducationSeptember 2008-Present

?Nanjing, China ?

?

Research Experience

?February 2011-Present

Research Projects:

? The modification of the antifungal Fluconazole;

? Synthesis of carbohydrate compounds containing β-amino alcohol via Henry

Reaction;

? The synthesis and applications of oxazolidinone compounds.Main Responsibility:

? Search literature, optimize experimental methods under the guidance;

? Carry out reactions and purify the products.?March-December 2010

1.Research Projects:

? Design and Applications of Functional Macrocyclic Compounds(College

Students' Science and Technology Innovation Training Projects of Jiangsu Province

(No.xxx and No.xxxx))

2.Chinese Patent:

? The Synthesis and Applications of Macrocyclic Compounds Containing The

Structure of Arylamine(Application No.xxxxxxxxx.x).3.Publication

? Synthesis and characterization of the novel shape-persistent nanosized cavity

macrocycle,synthetic metals 2011, XXX, XXXX-XXXX.Research Interests

? Synthetic organic chemistry in natural products;

? Asymmetric synthesis.English Proficiency

? IELTS: 6.5(Listening 6.5, Reading 7.5, Writing 6, Speaking 6.5)

Professional Skills

Chemistry

? Proficient in literature review, spectrum analysis and normal laboratory techniques;? Enable to conduct reactions with air sensitive reagents using a double manifold;? Monitor reactions with TLC, HPLC, GC;

? Purify and separate products using column chromatography.Computer

? Specialized software: ChemBio Office, Origin, Endnote, etc.;

? Literature searching online.Extracurricular Activities and Awards

?

?

?

?

?

President Scholarship, NUIST, Oct.2011 Award of Pacemaker to Merit Student, NUIST, Oct.2011 Award of Excellent Student Cadre, NUIST, Oct.2011 Pei Zhi Scholarship, NUIST, Jan.2011 First Prize in the 2nd Chemical Experiment & Skills Contest of NUIST, Jan.2011

Hobbies

? Badminton, Jogging, Reading, Music

第二篇：CV英文個人簡歷

personal Information:

Name: XXX

Sex: Male

Date of Birth: XX XXth XX

place of birth: XX, p.R.China

Marital Status: XX Health: Good

Mobile :13XXXXXXX

E-mail: wdjlw@www.tmdps.cn

Objective:

Application Engineer, Technical and Electronics Sales.Education Background:

2002.9??2006.6 XXXX.Bachelor of Electronic informationn engineering

Major subjects: Circuit principle, Single Chip Computer principle and Application, Digital Electronic Technology, Analog Electronic Technology, Interfacing and Application of Computer, principle of Communication,Work Experiences:

2006.7-present XXX As a computer software engineer, Responsibilites includes :

1.Solving environmental problems on prototype computers.Conducte systems analysis on new computer profucts to ensure hardware,software and mechanical design integrity

2.And responsible for System compatibility and integration test.Quickly diagnose causes of systems failures and malfunctions to ensure highest operating efficiencies,reliability,and quality performance standards.3.Assist engineers from Hp wIth installation and trobleshooting of Hp Bussiness pC software

English Skills

1.pass CET-4

2.Have a good command of spoken and written English.Communicate and negociate with

clients fluently in English.3.Skilled in reading documents relating to Electronics and Computer

Others

Independent and be able to work under pressue.Have coordination skills,teamwork spirit.Studious

nature and dedication are my greatest strengths

第三篇：(CV)英文個人簡歷正規格式

DENG XIAO PENGMobile: XXXXXX;Email: XXXXXXX

Name:

Gender: male

E-mail:

Tel:

Experience

2011-2012English intern in a poor mountain village elementary

school.2012-2013college student union.Manage the things that used in some big event.Education

2012.9-presentstudy in Wenzhou University, major in Business English.Main Courses American CultureHistory and Anthology of English LiteratureMicro-economyReading Course in American and British News Publications English-Chinese Translation The Art of Public Speaking A Survey: Major English-Speaking Countries Oral English Computer Skills

Strength

? English language skill: fluent in using English for work and communications

? Fast adaptation to new working environment and dedicated to efficient team

work

?

?

?

Strong experience in team building, project management and coordination.Project planning, budget control and manage deliverables on schedule.Business development and customer oriented communication.Interests

Playing music, badminton, chatting with friends, reading, English films,writing.

第四篇：出國留學申請CV

留學申請CV模板與范文

今天講CV先，之前收到小嘰同學的CV，打開一看，GPA旁邊還粘了一張用EXCEL做的GPA四年曲線表，把我昏過去了…所以決心先寫CV篇。CV，兩個字加三個符號：Be Professional！!一定要看上去專業，CV是你的門面，你的臉，所有的申請，讀書也好實習也好工作也好征婚也好，了解一個人，就憑這張紙，把自己化妝得漂亮點，關鍵是要讓人信得過，讓人覺得你靠譜，讓人覺得你professional！

我三年前申請的時候，也沒有任何人教，到處查CV的模板，自己反復改了很多遍改到煩死了，最后就遞交了算了，時間也來不及。

后來到了這里，學校有專門的就業指導中心，幫你改CV，各個學院也專門有針對各個學院的，我跑過很多個，也認真研習了各種學校提供的CV參考模板，有很系統的一套。我寫這個系列的目的就是指出CV寫作的要點，剖析常見錯誤。我以前犯過的，你們不要重蹈覆轍。

我每年都更新我的CV，主要是確實有需要的場合用到，第一年07年申請學校，第二年08年申請夏天的實習，第三年09年申請工作，后來又申請GA，針對每個工作內容都改一版新的。“針對”很重要！內容要針對！語言和排版要專業！就這兩個。

我為了寫這個CV篇重新翻出我當年申請時候的CV，和之后每年更改的CV，現在看當年的，簡直是相形見絀，哭笑不得，那幼稚的，拿不出手，遮臉遮臉。那么Pirate Radio（非常好看英倫搖滾電影 海盜電臺）里面說，能承認錯誤的，才稱得上是個男人。

話說，CV和RESUME，的區別，是RESUME就一頁，你人生之精華，就縮在一頁之內，是給人家一瞟眼了解你用的。

而CV，一個好習慣的且professional的人，是該每隔半年就更新一遍的，是你的編年史，每發生了一點變動，有新的內容，就補充上去，是長長的，細細的，是為了已經對你感興趣的人想更深入的了解你用的。比如RESUME里你一句話“2007年擔任iCow社團主席”

而CV里在這句話下面加五個標記點： “2007年擔任iCow社團主席：

-建立了全國第一家大學生有機奶牛農場；出席了2007國家牛奶生產加工會議；為社區以及大學學生生產了13加侖的有機牛奶；

參與擬定了第17項國家奶牛保護法”

我上面這樣寫不夠對仗，正式的簡歷，如果想好了以動詞開頭，每一個句子的第一個詞都要動詞開頭，如果是副詞“獨立擔當了”，下一個就要以“團隊形式地合作了”，要詞性對仗，懂？ 所以我以上應該改成： “ iCow社團主席 2007生產了13加侖的有機牛奶為社區以及大學學生；參與擬定了第17項國家奶牛保護法”

我把第二句的順序移動了，這樣人家 一瞟眼 RESUME的一掃，第一豎列，都是動詞，都是行為，句子都是動賓結構，這樣讓人家很容易看懂，你顛來倒去，人家只要有一個wonder，你到底在寫什 么，馬上心里的好感會降下來，馬上會引起心煩，那你present你的RESUME是要設想，是一個給教授 按摩的過程，把人家弄舒服，人家才愿意招你一起合作。

精細到如此精心的準備你的RESUME，才算合格。

BTW，以動賓開頭最好，action 的動詞很catch人家的眼球。

6.每一件事情發生的時間要標出來。簡歷上精確到季度或年份就可以了。美國這邊對時間很注意，隨便什么地方簽名旁邊都要標個時間，支票也是，發票也是，不要說一般的文件表格了。7.GPA盡量算成4分制。

美國按照4分制的, 國內有些學校按照5分的，換算成4分制，否則來個4.XX的GPA這里還要給你重新算，那你還不如最開始就用最能把自己的GPA算高的算法算出來呈現給對方呢

8.用逗號分隔句子，而不是用分號.分號在英語里的用法我到現在都吃不準，和中文里的分號用法是不一樣的。所以如果要分隔句子，用逗號。

Conducted research on…., managed team project xxx, and developed xxxx 而不是

Conducted reseach on…;managed team project xxx;developed xxxx 9.給沒有研究背景的非牛牛們再來一道法子: Course project

我在這里第一個學期讀研究生的時候，才讀了幾個月，實習的招聘會就開始了。我都還沒學什么呢，更表說研究背景了。我去就業中心問這個簡歷要怎么寫 寫，我剛來，都沒這個專業的背景。對方說，不要慌，大部分學生都沒研究背景的，把你學的課程列出來，你的課業里的作業列出來，就把那些作業作為你的 project experience。

美國這里的課程作業，做項目的多，有的是團隊項目，有的是個人的，一般研究生都是獨立的項目。比如我做過一個要去學生宿舍樓里檢測安裝設備的，還有 一個是大樓的險情狀況計算機模擬，一個項目一般給一個月的時間，從文獻綜述，編寫程序或者出訪調查，到寫份幾十頁的報告，到PPT演講。這一類的作業很練 人。做完了就真的懂很多。就業中心的指導說，把這類作業寫到你的簡歷里去，比如你的一門作業，老師布置，每個學生采集一頭奶牛的奶水樣本，你在簡歷上就 寫：

-獨立采集大學有機奶牛農場牛奶樣本 寫得好聽一點呢，可以說：

-在大學有機奶牛農場做牛奶生產檢測

很多學工科的學生，大多有些實驗課，把這些課的內容寫出來：-研究了17種傳感器的工作原理，包括…… 社會學科和商科大多有調查課題：

-調查了17頭奶牛哺乳期的心理活動，對此撰寫了一份17頁的英文調查報告，并正式演講了調查發現(formally presented the results / findings / it)就業指導說，大家都是學生，就是沒有什么特別尖銳的學術研究背景的，就把課堂里的作業寫出來，真的一個一個去想去列做過哪些作業，認真的漂亮的措詞寫到CV上，就會變成一份很豐富的簡歷，人家一看你做過很多事情。簡歷好不好看的關鍵在于你如何present你的過去。

本來么，你確實做過很多事情，你確實在某門課的作業里設計了某個電路，確實寫了十篇實驗報告，為什么不擺出來說呢？

不要以為只有研究生的或者跟著導師發論文的研究才算得上研究背景，你讀了4年大學，每個人的軌跡不一樣，別人花很多時間在實驗室里，你必定把這些時 間花在了別的事情上，除了逛街買衣服磨臉打游戲，你比別人多做的有什么，如何安排你的時間，4年時間的一個專業的學習，肯定是做過不少跟專業相關的事情 的。

對方的招生或者教授，看的也就是你： a.有沒有做研究的能力 b.有沒有寫論文的的能力 c.有沒有做演講的能力

你如果在一門實驗課里做了10個實驗，那就是有很好的動手能力，把它拿出來說呀，這些經歷無非不是在某個特別的導師的辦公室里做的而已，就是課堂做 的，怎樣，我要體現的就是，我懂電路，我懂編程，我懂模擬，我懂傳感，我懂數學模型……你要體現的不就是你有這個能力么？糾結于它是在哪里完成的干嘛？

我以前也笨的，明明課堂作業里做過很多小測試小project，但總覺得那是作業啊，那不算什么啊，那人人都做的作業啊，不是高深的研究啊。所以當時一個都沒放，只覺得專業背景空空。

其實是錯的，明明做過很多跟專業相關的事情，你跟導師研究室里做出來的東西，無非只是課堂實驗室的延長版，一樣的本質啊。

所以，每個人都必定有起碼5行的專業研究背景可以寫，請打印出你們的成績單一門一門課去想，在那門課里做過了什么吧。

我當初到了這里第一學期才讀了幾個月的課，一共三門課，要我寫簡歷，我就把三門課里做的project一個一個列寫出來，一門課里有三個project，另一門課里有7個實驗，另一門課里有一份大報告，就這樣整整列了一整頁的簡歷，簡歷就滿滿的很有內容很漂亮了。

他招生單位怎么知道你在學校每門課的老師都布置什么作業？他招的來自這么多學校的怎么會知道你們學校什么課程設置和要求？ 他要看到的就是你有經驗，有能力。你要呈現的也就是“我能”。簡歷的關鍵在于如何呈現。

不要說你想想自己一片空白，過去沒做什么。

給我認真冥想，仔細列出來。豐滿你的簡歷吧 10.用數字說話。

小嘰給我的簡歷里有一項她組織文藝活動的內容，寫 “Organized many musical parties”

我讀下去，知道她寫的是她組織過很多活動

首先“parties”這個詞在美國多指瘋狂小本們的周末酒精派對，用詞不準，用events更合適

再，many 這個詞不好，沒有力道，這個時候就要用數字，改成

“Organized more than twenty campus-wide events including two annual chorus contests, three New Year Gala, …., etc.” 11.想用引號的時候，用斜體而不是引號。

這里也有個標點問題。中文里用引號的地方，英文里往往是用斜體。比如： 我的研究課題是“天花板下的母奶牛” 在英文簡歷里改成

我的研究課題是 天花板下的母奶牛

研究課題啦，三好學生 之類的獎學金稱號啦，書名啦，最好都改成斜體，而不是引號，比較符合英語習慣。我到現在都不知道雙引號在英文里怎么用的，很少看到。

簡歷中表示強調沒有什么死規定，粗體，斜體，下劃線，自己安排。保持前后一致，不要太瘋狂就好。

沒有錯，雙引號和斜體應當都可以。

而且在參考文獻里，對期刊，會議論文等引用格式不同，有一種用的是雙引號沒錯。

但我的日常接觸中，除了引用別人說話的內容用雙引號，其他地方很少見。尤其是在非參考文獻的場合，比如一本書里作者提及另一本書的標題，在我以往接觸到的資料里，往往常見的是斜體，而非引號。

簡歷中提及自己畢業設計的名字，或者課程項目的名字，不是正規的已經發表了的論文，我認為 斜體 較 引號 更為妥當。

第五篇：申請國外大學博士的計劃書

Research Interest Proposal I am the Doctor degree applicant Xu Zhiwei.From September 2008 to June 2013, I studied the laboratory medicine at Nantong University.In September 2013 I was recommended to the graduate school by the professors due to my excellent assessment.Through undergraduate studies, I contacted such theories as molecular biology, cellular biology , immunology and so on.However, keenly conscious that my current acquisition is far from enough for me to meet the needs of the fast developing medicine, I am eager to pursue further studies in Chinese medical sciences.With my hard working, I know about the basic knowledge of medical immunology.Up to now, I have known a lot about the University of Macau.The graduate school of university plays a prominent role in the academic research and transforms Macao into a knowledge-based society.I am dedicated to pursuiting medical research and hoping to come to the university of Macau for rich experiences and success in PhD program.I am now applying for Chinese Medical Science in the University.This planned research has the following aims: The deubiquitinating enzyme USP14 has been identified and biochemically studied, but its mechanisms in cancer remains to be elucidated.Protein glycosylation with O-linked N-acetylglucosamine(O-GlcNAc)is a reversible post-translational modification occurring onserine or threonine residues.Intriguingly, it has been observedthat O-GlcNAcylation is particularly abundant on cancer cells.The aim of this study was to evaluate the O-GlcNAcylation of USP14 in patients with cancer and to define its mechanisms in cancer cell proliferation and apoptosis.a.To demonstrate that O-GlcNAcylation of USP14.b.To explore Interplay between USP14 O-GlcNAcylation and phosphorylation c.To determine

how

O-GlcNAcylation

regulates

metabolic reprogrammingand Signaling in cancer cells.d.To test whether O-GlcNAcylation regulates proliferation and apoptosis.2.Research Context

Deregulating cellular energetics is emerging as a characteristichallmark of cancer cells.Withinsuch cells, glucose and glutamine are used at an increasedrate, resulting in the production ofATP in a manner independent of oxygen concentration.Elevated glucose and glutamine flux areneeded not only to serve the energetic demands of cancer cells,but also to provide the essential carbon and nitrogen used inmacromolecule synthesis, fueling the rapid growth and proliferation seen in tumors.This increasein glucose and glutamine uptake can alter multiple metabolicand signaling pathways in cancer cells, including for examplethe hexosamine biosynthetic pathway(HBP).The HBP relies on glucose and glutamine uptake, and approximately 3%–5% of the total glucose entering a cell is shunted intothis pathway.This critical metabolite isrequired for the biosynthesis of a variety of extracellular glycopolymers, including both N-and O-glycans, however, it also serves as the substrate for O-linked b-N-acetlyglucosamine

(O-GlcNAc)

transferase

(OGT).O-GlcNAcylation is directly involved in growth hormone(gibberellic acid)signalling in plants, and both SPY and SECRET AGENT(SEC)encode O-GlcNAc transferases.Mutations in either SPY or SECcause severe growth defects;simultaneous mutation of both genes islethal.Unlike plants, mammals and insects seem to have only a single gene encoding the catalytic subunit of the O-GlcNAc transferase(OGT)Gene disruption in

mice

established

that

OGT

is

required forembryonic-stem-cell viability20.Tissue-targeted disruption in miceshowed that O-GlcNAcylation is essential to several cell types.OGTdeletion causes hyperphosphorylation, which is followed by cell death, induces T-cell apoptosis and causes growth arrest infibro blasts.Cre–lox-mediated deletion of OGT in cultured fibroblastsresults in death as pre-existing OGT protein levels diminish.This modification can be removed by theglycoside hydrolase O-GlcNAcase(OGA)that catalyzes cleavage of O-GlcNAc from proteins.This modification can alter protein functiondirectly or, in some cases, by competing with phosphorylationsites.O-GlcNAc and O-phosphate site-mapping studies suggest that there areat least four different types of dynamic interplay between O-GlcNAcand O-phosphate.First, there is competitive occupancy at thesame site, for example that which occurs in the transcription factorc-Myc25 and oestrogen receptor-β26, and on the oncoprotein SV-40 largeT-antigen27 and endothelial nitric oxide synthase28.Second, competitiveand alternative occupancy occur at adjacent sites, such as that observedin the tumour suppressor p53 and synapsin I.Third,there is a complex interplay whereby some O-phosphate attachmentsites on a given protein are the same as some O-GlcNAc sites, whereasothers are adjacent to, or even distant from, each other, such as on theC-terminal domain of RNA polymerase II and on cytokeratins32.The final type of interplay involves proteins in which this relationship has yet to be clearly defined.The interplay between O-GlcNAcand O-phosphate is also underscored by the recent finding that OGTtransiently forms complexes containing the catalytic subunit of proteinphosphatase 1(PP1c).Cancer cells, however, uptake glucose at a higher rateand produce lactic acid rather than metabolizing pyruvate throughthe tricarboxylic acid(TCA)cycle.This adaptive metabolic shift is termed the Warburg effect, leading to anaerobic glycolysis, and is thought toprovide an evolutionary advantage to cancer cells by providingboth increase bioenergetics and biosynthesis.Many protooncogenes(e.g., Ras and Myc)and tumor suppressors(e.g., p53)influence metabolism,and mutations in these genes can upregulateglucose uptake in cancer cells and promote a metabolic phenotype supporting

tumor

cell

growth

and

proliferation.Elevatedglucose uptake in cancer cells can be applied to monitor the location of primary and metastatic tumor sites;for an example, usingF-18 fluorodeoxyglucose(FDG), a glucose analog, with a combination of positron emission tomography/computed tomography(PET/CT).Recent study has providedinsights into the mechanism ofpost-translational modification of molecules in cancer cells theregulation of many molecules and

suggested

important

implications

in

cancer development.Additionally, lines ofevidence of global proteomic analysis havesuggested that post-translational modifications of USP14 are likely not limited to phosphorylation.Other forms of modifications, such asO-GlcNAcylationappear to occur as well,suggesting a more sophisticated regulatorynetwork of USP14.In this study, we would evidence that O-GlcNAcylation within cancer cells regulates cancer cell metabolism via regulation of phosphorylation and its downstream target genes.Mechanistically, we wonder which signaling pathway has participated in the regulation.Furthermore, we will discuss whether decreased O-GlcNAcylation leads to reduced proliferation and apoptosis incancer cells.In addition, we hypothesized that human cancers containing high USP14 levels also containelevated OGT and O-GlcNAcylation.Importantly, we will explore in overallcancer patients, lower OGA expression correlates withpoor clinical outcome.Thus,we will confirm that O-GlcNAcylation serves as a criticallink between the key pathways thatare critical for cancer cell survival via regulation of glycosylation.Method: a.To demonstrate that O-GlcNAcylation of USP14.Here, the O-GlcNAc moietyon the protein is labelled with UDP-GalNAz using a mutantgalactosyltransferase GalT1 Y289L(mGalT1)with an azidederivative of UDP-GalNAc(UDP-GalNAz)as donor substrate,followed by labelling with biotin alkyne.After in vitro O-GlcNAcylation, USP14 was subjectedto mGalT1 labelling and then detected by probing withstreptavidin-conjugated HRP.b.To explore Interplay between USP14 O-GlcNAcylation and phosphorylation.Wewill explore that whether increasing USP14 O-GlcNAc modification with GlcN orPUGNAc treatment inhibits NF-κB activation and have delineatedthe molecular mechanisms of this effect.We will demonstrate that USP14 is a target for O-GlcNAc modification inGlcN or PUGNAc treated cells, and that this post translationalmodification prevents its phosphorylation in response to TNF-α,suggesting a reciprocal relationship between O-GlcNAcylation andphosphorylation of USP14 in cancer cells.We would further showthat, in cancer cells pretreated with GlcN or PUGNAc, levels of O-GlcNAcylation and phosphorylation of USP14 was changed.c.To determine

how

O-GlcNAcylation

regulates

metabolic reprogrammingand Signaling in cancer cells.Since OGT and O-GlcNAc has been associated with regulationof metabolic diseases such as insulin resistance, we hypothesized that OGT could serve as an importantregulator of glycolytic metabolism to regulate cancer cell growth.To test this idea, we initially examined the effect of OGT reduction on metabolites from human cancercells using liquid chromatography-mass spectrometry(LC-MS).The metabolic profile of cancer cellscontaining OGT knockdown with RNAi demonstrated a generaldecrease in glycolytic and pentose phosphate pathway(PPP)metabolites and an increase in tricarboxylic acid(TCA)cycle metabolites, consistent with a reversalof the Warburg effect and inhibition of cancer cell growth underthese conditions that we and othershave previously shown.d.To test whether O-GlcNAcylation regulates proliferation and apoptosis.Glucose deprivation and antiglycolytic drugs can selectivelyinduce tumor cell proliferation and death;thus, we examined the effect of reducing O-GlcNAcylation on proliferation and apoptosis in nontransformed immortalized mammary epithelial cells compared tocancer cells.In cancer cells stablyexpressing control siRNA or OGT siRNA at day 8 post-infection,we we tested whether cell rounding and detachment of cancer cells containing OGT knockdown, while cancer cells attached to the plate.To further investigate the effect of OGT SiRNA on cellular proliferation, we used chemically synthesized siRNA to knockdown endogenous OGT in cancer cells.The efficiency of the OGT-targeted siRNA-mediateddown-regulation was assessed by Western blot analysis.Aspredicted, siRNA knocked down the protein expression of OGT as compared with negative control siRNA and mocktreatment.To determine the effect of OGT knockout on cancer cell proliferation, OGT-siRNA, negative control-siRNA and mock treatment cancer cell proteins were testedby Western blot.We would explore that expressed decreased OGT levels hadelevated cleaved caspase-3 and caspase-8, and upto 50%–70% of cells were examined for annexin V.