第一篇：英語醫藥學專業

1.Introduction to quantitative risk assessment

2.Risk analysis is a valuable tool in the management of

microbial food safety issues and can provide a systematic

approach for the regulatory authorities and the food industry

to control the risk posed by a pathogen in a particular

food commodity.Risk analysis consists of three elements:

risk assessment, risk management and risk communication.Risk assessment is the scientific part of the process in which

the hazards are identified and the risk posed by that particular

hazard(i.e.pathogen)is calculated.The principles of

risk assessment including the four stages involved(hazard

identification, exposure assessment, hazard characterisation

and risk characterisation)are outlined by the Codex

Alimentarius Commission(Codex, 1999).Each of the stages is summarised below.1.1.Hazard identification

A hazard is defined as an agent having an adverse effect

on the public health of the human population and may

pose a short term, chronic, or fatal risk to a person.The

identification of microbial hazard associated with a particular

food is generally based on information generated from

routine microbial analysis of the commodity or from an

epidemiological linkage of a particular pathogen with a

case of food borne infection.1.2.Exposure assessment

Exposure assessment is a quantitative estimation of the

presence of a contaminant in a serving of food at the time

of consumption, or as close to this stage as is scientifically

possible and practical.However, the final estimation of the numbers and prevalence of a pathogen in the food is of ten

based on an accumulation of data on the prevalence and

numbers of pathogen at key points in the food chain with

data included on how particular stages in the food chain

affect the numbers/prevalence of the pathogen.The final

step in the process estimates the amount of contaminant

in a single serving, with information on the typical amount

of food consumed in a serving procured from nutritional

Databases.The exposure assessment model can be ‘deterministic’,i.e.derived using single data points along the food chain.However, this approach may result in outlier values being

ignored and thus under or overestimating the risk.A more

common approach is to use a probablistic or stochastic

analysis, in which a distribution curve representing all data

is used as opposed to a single point estimate.Typically a

Monte Carlo analysis is used to include data from all the

distributions along the chain and is done using software

such as @Risk(Palisade, NY, USA).In these analyses, a

single data point is chosen at random from each distribution

curve and used to calculate an outcome.The process

is repeated several thousand times(multiple iterations)with

a different data point in each distribution chosen each time

and with the final output being based on all the iterations.The error in the predicted risk may be due to variability or

uncertainty, and there is increasing emphasis being placed

on quantifying and separating the impact of both uncertainty

and variability in risk assessments(Cohen, Lampson,& Bowers, 1996;Pouillot, Beaudeau, Denis, &

Derouin, 2004).1.3.Hazard characterisation

Hazard characterisation relates exposure to a hazard

with the probable public health outcome(illness/death).A

dose–response relationship can be used to estimate the

amount(number)of pathogens which causes illness.The

data used in generating dose–response models are derived

from a variety of sources including human clinical trials,epidemiological studies based on food poisoning outbreaks,animal clinical trials, in vitro studies using cell lines,biomarkers or expert opinion.In some cases, the dose–

responses will describe the susceptibility of different populations,i.e.general population and immunocompromised.1.4.Risk characterisation

The final stage in the process estimates the adverse

public health effect, or risk as a consequence of exposure

to the hazard.This may be a prediction of illness per typical

serving or calculated as an annual risk of illness.Depending on the hazard characterisation data available,the risk estimates may be broken down into age categories,based on differences in immune status in order to

identify groups which may be at higher risk following

exposure to the contaminant.The risk characterisation

model is generally developed using commercial software such as @Risk or Crystal Ball(Decisioneering Inc., Denver,USA).These programs can separate the distribution

for the overall risk prediction into uncertainty and variability

to allow more complex risk estimation and analyses

of the data.The generated model can be used to assess

which parts of the chain significantly affect risk or to

assess the changes in predicted illness by incorporation

of a new hypothetical risk mitigation strategy at a particular

point in the chain.This paper reviews Escherichia coli O157:H7 in the farm

to fork beef chain and examines how quantitative risk

assessment models have been applied to establish and manage

the risk posed.While other serovars of verocytotoxigenic

E.coli(including E.coli O26, O111, O103, O145)

are now emerging as a cause of similar illness to E.coli

O157:H7 they are not addressed in this paper as there is

still limited information on their transmission thorough

the beef chain and they have not been included in any published

quantitative risk assessment models.2.E.coli O157:H7: human clinical aspects

E.coli O157 is a member of the Enterhaemorrhagic

group of E.coli(EHEC)and was first implicated in infectious

disease in the early 1980s(Riley et al., 1983).The

symptoms of infection include bloody diarrhoea and severe

abdominal pain.Haemolytic uraemic syndrome(HUS), a

cause of acute renal failure, may be a complication of the

illness, and neurological problems in the form of thrombotic

thrombocytopaenic purpura(TTP)may also occur.Immuno-compromised patients, including young children

and the elderly, are at particular risk of developing HUS.The time from exposure to onset of symptoms ranges fromto 14 days(Coia, 1998).However, with complications the

illness may last many months and lead to permanent damage

or even death.Pathogenicity is related to the ability of

the organism to adhere to and colonise the human large

intestinal epithelial tissue, forming attachment and effacing

(AE)lesions and the production of verocytotoxins.The

E.coli verocytotoxins are closely related to the Shiga toxin

of Shigella dysenteriae and are typically bacteriophage

encoded.There are two main classes of verotoxin: VT1, a

homogeneous group of toxins, virtually identical to the

Shiga toxin of Shigella and VT2, a heterogeneous group

of toxins, more distantly related to the Shiga toxin.E.coli O157 with the eae gene and VT2 are most often

associated with HUS in patients(Werber et al., 2003).Outbreaks of VTEC infections involving serovar O157

have now been reported from United States and Canada

Bell et al.(1994)(Lisbea), Asia(Michino et al., 1998), Australia

(Desmarchelier, 1996), Europe(Tozzi, Gorietti, &

Caprioli, 2001), and Africa(Germani, Soro, Vohito,Morel, & Morvan, 1997).However, the majority of cases

are sporadic and contribute significantly to overall cases

of infection.There is considerable variation in infection

rates between different geographical regions.In Europe, the

highest rates of infection are in Scotland with approximately 4 cases per 100,000(SCIEH, 2006).In the Republic of Ireland

the incidence per 100,000 has ranged from a peak of

2.2 in 2003 to 1.3 in 2004(HPSC, 2004).In Northern Europe

infection rates are very low ranging from 0.04 per

100,000 in Norway and Finland to 1.1 in Denmark in

2000 although Denmark has in 2006, reported its first general

outbreak of E.coli O157 attributed to contaminated

milk(Jensen et al., 2006).In 2004, the incidence rate for

E.coli O157:H7 in North America was 0.9, a drop from

1.1 cases in 2003.In Asia, Japan has experienced the most

problems related to E.coli O157:H7 with an average incidence

rate of 2.74 per 100,000 between 1999 and 2004

(Sakuma, Urashima, & Okabe, 2006).A number of sources

and reservoirs of E.coli O157 including beef and lamb,lettuce, sprouts, fruit juices, vegetables, raw milk, water

have been implicated as vehicles of transmission(Bell

et al., 1994;Cowden, Ahmed, Donaghy, & Riley, 2001;

Hilborn et al., 2000;Michino et al., 1999).Person-to-person

is also an important mode of transmission, particularly

in day care centers(O’Donnell et al., 2002)and direct contact

with animals carrying the organism or with faecally

contaminated mud(Anon, 1999;Crampin et al., 1999)

are also recognised sources of infection

第二篇：醫藥學專業自我介紹

醫藥學專業自我介紹(精選多篇)

好范文為大家整理了以下這一份關于醫藥學專業畢業生的自我鑒定范文，僅供廣大畢業生前來參考一下。

由于經過一個學期的學習，我知道了醫學理論學作為醫學與理論學相交叉的邊緣學科，其宗旨在于提高學生的醫學人文素質和綜合職業素質，再加上后來的實踐活動使理論更加與實際的緊密聯系，令我認為學習醫學理論學成為醫學生一門必須學習的課程。

在“藥學中西、醫學濟世”八字校風的鞭策下，我努力學習，刻苦鉆研、勇于進取，時刻向“將自己培養成為具備高綜合素質的臨床藥學畢業生”的目標奮進。我還獲得了學校三好學生和二等獎

學金等重要獎項。學習當中我深深的體會到，我們以履行公民義務為光榮，本著社會共濟、關愛他人的精神，用愛心共同托起生命的希望。血液是生命的源泉，愛是生命的曙光。生命之源聯系著你、我、他，我們的愛心是無限的。

所以在有限的學習期間，我在學校形成尊重勞動、尊重知識，培養德、智、體、美全面發展的高素質學生，注重學術的理念：崇尚學術，營造發揚學術民主和學術自由、重視學術成就的濃郁學術氛圍。只有堅持這種理念，才能不斷取得科學研究的豐碩成果，才能不斷提高自身的學術水平和知識質量，知識創新和文化傳播等做出應有貢獻。

花蕾要綻放，不是在溫室，而是在肥沃的土壤上吸收天地日月精華，經受風霜雨雪考驗。我要成才，我必須在廣闊天地里自我歷練，真正在熟悉自我、完善自我、熟悉社會、服務社會的社會實踐中成長為社會英才。只有熟悉了自我，完善了自我，才能更好地熟悉社會，服務社會;只有在熟悉社會、服務社會的過程里才能更好地熟悉自我、完善自我。

在往后的學習中，我會更加努力，我會牢記著醫學生的誓詞：我自愿獻身醫藥學，熱愛祖國，忠于人民，恪守藥德，尊師守紀，刻苦鉆研，孜孜不倦，精益求精，面發展。我決心竭盡全力除人類之病痛，助健康之完美，維護醫術的圣潔和榮譽，救死扶傷，不辭艱辛，執著追求，為祖國醫藥衛生事業的發展和人類身心健康奮斗終生。

下面就一起來欣賞以下這一份關于醫藥學專業學習的自我鑒定范文，歡迎大家瀏覽。

由于經過一個學期的學習，我知道了醫學理論學作為醫學與理論學相交叉的邊緣學科，其宗旨在于提高學生的醫學人文素質和綜合職業素質，再加上后來的實踐活動使理論更加與實際的緊密聯系，令我認為學習醫學理論學成為醫學生一門必須學習的課程。

在“藥學中西、醫學濟世”八字校風 的鞭策下，我努力學習，刻苦鉆研、勇于進取，時刻向“將自己培養成為具備高綜合素質的臨床藥學畢業生”的目標奮進。我還獲得了學校三好學生和二等獎學金等重要獎項。學習當中我深深的體會到，我們以履行公民義務為光榮，本著社會共濟、關愛他人的精神，用愛心共同托起生命的希望。血液是生命的源泉，愛是生命的曙光。生命之源聯系著你、我、他，我們的愛心是無限的。

所以在有限的學習期間，我在學校形成尊重勞動、尊重知識，培養德、智、體、美全面發展的高素質學生，注重學術的理念：崇尚學術，營造發揚學術民主和學術自由、重視學術成就的濃郁學術氛圍。只有堅持這種理念，才能不斷取得科學研究的豐碩成果，才能不斷提高自身的學術水平和知識質量，知識創新和文化傳播等做出應有貢獻。

花蕾要綻放，不是在溫室，而是在肥沃的土壤上吸收天地日月精華，經受風霜雨雪考驗。我要成才，我必須在廣

闊天地里自我歷練，真正在熟悉自我、完善自我、熟悉社會、服務社會的社會實踐中成長為社會英才。只有熟悉了自我，完善了自我，才能更好地熟悉社會，服務社會;只有在熟悉社會、服務社會的過程里才能更好地熟悉自我、完善自我。

在往后的學習中，我會更加努力，我會牢記著醫學生的誓詞：我自愿獻身醫藥學，熱愛祖國，忠于人民，恪守藥德，尊師守紀，刻苦鉆研，孜孜不倦，精益求精，面發展。我決心竭盡全力除人類之病痛，助健康之完美，維護醫術的圣潔和榮譽，救死扶傷，不辭艱辛，執著追求，為祖國醫藥衛生事業的發展和人類身心健康奮斗終生。

下面就一起來欣賞以下這一份關于醫藥學本科畢業生的優秀自我評價范文，歡迎廣大畢業生瀏覽。

驀然回首四年大學生活，當年單純懵懂的少年已成成熟穩重之人，使我有此巨變的正是那段不凡的人生經歷以及其對夢想堅持不懈的努力。

本人努力學習，刻苦鉆研、勇于進取。在四年里，曾當任過班長、學生會學習部部委、學生社團聯合會文化部部長等校內重要學生干部，曾多次參加過大量的校內外的活動，由于成績突出，本人還獲得了學校三好學生和二等獎學金等重要獎項。尊敬老師，團結同學，在校內擁有廣泛的群眾基礎。

在兼顧學業的前提下，還不忘對自身能力的培養，積極參加各種校內校外的培訓，拓寬了眼界的同時，積累了大量的社會實踐經驗，使德智體得到全面的發展。在實習期間，持著主動求學的學習態度，我積極向帶教老師學習，秉著“健康所系性命相托”的信念，孜孜不倦地吸收醫藥學知識，為日后的學習、工作打下堅實的基礎。由于工作認真，表現出色，得到科室的一致好評。

我將在以后的工作和學習中更加努力，不斷充實自我、完善自我，刻苦鉆研，孜孜不倦，精益求精，竭盡全力除人類之病痛，為祖國醫藥衛生事業的

發展和人類身心健康奮斗終生。

1.introduction to quantitative risk assessment

2.risk analysis is a valuable tool in the management of

microbial food safety issues and can provide a systematic

approach for the regulatory authorities and the food industry

to control the risk posed by a pathogen in a particular

food commodity.risk analysis consists of three elements:

risk assessment, risk management and risk communication.risk assessment is the scientific part of the process in which

the hazards are identified and the risk posed by that particular

hazard is calculated.the principles of

risk assessment including the four stages involved are outlined by the codex

alimentarius commission.each of the stages is summarised below.1.1.hazard identification

a hazard is defined as an agent having an adverse effect

on the public health of the human population and may

pose a short term, chronic, or fatal risk to a person.the

identification of microbial hazard associated with a particular

food is generally based on information generated from

routine microbial analysis of the commodity or from an

epidemiological linkage of a particular pathogen with a

case of food borne infection.1.2.exposure assessment

exposure assessment is a quantitative estimation of the

presence of a contaminant in a serving of food at the time

of consumption, or as close to this stage as is scientifically

possible and practical.however, the final estimation of the numbers and prevalence of a pathogen in the food is of ten

based on an accumulation of data on the prevalence and

numbers of pathogen at key points in the food chain with

data included on how particular stages in the food chain

affect the numbers/prevalence of the pathogen.the final

step in the process estimates the amount of contaminant

in a single serving, with information on the typical amount

of food consumed in a serving procured from nutritional

databases.the exposure assessment model can be ‘deterministic’，i.e.derived using single data points along the food chain.however, this approach may result in outlier values being

ignored and thus under or overestimating the risk.a more

common approach is to use a probablistic or stochastic

analysis, in which a distribution curve representing all data

is used as opposed to a single point estimate.typically a

monte carlo analysis is used to include data from all the

distributions along the chain and is done using software

such as @risk.in these analyses, a

single data point is chosen at random from each distribution

curve and used to calculate an outcome.the process

is repeated several thousand times with

a different data point in each distribution chosen each time

and with the final output being based on all the iterations.the error in the predicted risk may be due to variability or

uncertainty, and there is increasing emphasis being placed

on quantifying and separating the impact of both uncertainty

and variability in risk assessments.1.3.hazard characterisation

hazard characterisation relates exposure to a hazard

with the probable public health outcome.a

dose–response relationship can be used to estimate the

amount of pathogens which causes illness.the

data used in generating dose–response models are derived

from a variety of sources including human clinical trials，epidemiological studies based on food poisoning outbreaks，animal clinical trials, in vitro studies using cell lines，biomarkers or expert opinion.in some cases, the dose–

responses will describe the susceptibility of different populations，i.e.general population and immunocompromised.1.4.risk characterisation

the final stage in the process estimates the adverse

public health effect, or risk as a

consequence of exposure

to the hazard.this may be a prediction of illness per typical

serving or calculated as an annual risk of illness.depending on the hazard characterisation data available，the risk estimates may be broken down into age categories，based on differences in immune status in order to

identify groups which may be at higher risk following

exposure to the contaminant.the risk characterisation

model is generally developed using commercial software such as @risk or crystal ball.these programs can separate the distribution

for the overall risk prediction into uncertainty and variability

to allow more complex risk

estimation and analyses

of the data.the generated model can be used to assess

which parts of the chain significantly affect risk or to

assess the changes in predicted illness by incorporation

of a new hypothetical risk mitigation strategy at a particular

point in the chain.this paper reviews escherichia coli o157:h7 in the farm

to fork beef chain and examines how quantitative risk

assessment models have been applied to establish and manage

the risk posed.while other serovars of verocytotoxigenic

e.coli

are now emerging as a cause of similar illness to e.coli

o157:h7 they are not addressed in

this paper as there is

still limited information on their transmission thorough

the beef chain and they have not been included in any published

quantitative risk assessment models.2.e.coli o157:h7: human clinical aspects

e.coli o157 is a member of the enterhaemorrhagic

group of e.coli and was first implicated in infectious

disease in the early 1980s.the

symptoms of infection include bloody diarrhoea and severe

abdominal pain.haemolytic uraemic syndrome , a

cause of acute renal failure, may be a complication of the

illness, and neurological problems in the form of thrombotic

thrombocytopaenic purpura may

also occur.immuno-compromised patients, including young children

and the elderly, are at particular risk of developing hus.the time from exposure to onset of symptoms ranges from to 14 days.however, with complications the

illness may last many months and lead to permanent damage

or even death.pathogenicity is related to the ability of

the organism to adhere to and colonise the human large

intestinal epithelial tissue, forming attachment and effacing

lesions and the production of verocytotoxins.the

e.coli verocytotoxins are closely related to the shiga toxin

of shigella dysenteriae and are

typically bacteriophage

encoded.there are two main classes of verotoxin: vt1, a

homogeneous group of toxins, virtually identical to the

shiga toxin of shigella and vt2, a heterogeneous group

of toxins, more distantly related to the shiga toxin.e.coli o157 with the eae gene and vt2 are most often

associated with hus in patients.outbreaks of vtec infections involving serovar o157

have now been reported from united states and canada

bell et al., asia , australia , europe , and africa.however, the majority of cases

are sporadic and contribute significantly to overall cases

of infection.there is considerable

variation in infection

rates between different geographical regions.in europe, the

highest rates of infection are in scotland with approximately 4 cases per 100,000.in the republic of ireland

the incidence per 100,000 has ranged from a peak of

2.2 in 2014 to 1.3 in 2014.in northern europe

infection rates are very low ranging from 0.04 per

100,000 in norway and finland to 1.1 in denmark in

2014 although denmark has in 2014, reported its first general

outbreak of e.coli o157 attributed to contaminated

milk.in 2014, the incidence rate for

e.coli o157:h7 in north america was 0.9, a drop from

1.1 cases in 2014.in asia, japan has experienced the most

problems related to e.coli o157:h7 with an average incidence

rate of 2.74 per 100,000 between 1999 and 2014

.a number of sources

and reservoirs of e.coli o157 including beef and lamb，lettuce, sprouts, fruit juices, vegetables, raw milk, water

have been implicated as vehicles of transmission.person-to-person

is also an important mode of transmission, particularly

in day care centers and direct contact

with animals carrying the organism or with faecally

contaminated mud

are also recognised sources of infection

二、名詞術語

醫學及藥學名詞應使用全國自然科學名詞審定委員會公布的規范名詞為準。

1.現將常易出錯的不規范名詞糾正如下。

氨基酸轉移酶細胞紅細胞

作用機制側支循環綜合征

膽固醇單核－吞噬細胞系統

低鉀血癥高脂血癥內鏡

固醇發紺反胃

分枝桿菌肺源性肺梗死

腦出血腦梗死心肌梗死

腦卒中放射性核素功能

肝硬化核糖體晶狀體

膠原纖維假膜咳痰

咯血抗生素磷脂酰膽堿

黏膜清蛋白期前收縮

妊娠高血壓綜合征三酰甘油

腎衰竭食欲缺乏食管

嗜酸性細胞性白細胞）同工酶

糖原畏食糖皮質激素

圍生期下丘腦心排出量

心源性血紅蛋白血流動力學 藥源性醫源性真菌 原發性高血壓

第三篇：大學生職業生涯規劃書醫藥學專業

大學生職業生涯規劃書醫藥學專業

gkstk小編為大家搜集了一篇關于醫藥學專業大學生職業生涯規劃書，供大家參考借鑒。

一、藥學專業環境及評價

藥學專業的就業方向十分廣闊，與藥品相關的各個領域(主要包括藥品研究開發部門、生產部門、管理部門、營銷及使用部門)都需要藥學專業的畢業生。具體而言有醫院、科研院所、藥廠、醫藥藥學專業的就業方向十分廣闊，與藥品相關的各個領域(主要包括藥品研究開發部門、生產部門、管理部門、營銷及使用部門)都需要藥學專業的畢業生。具體而言有醫院、科研院所、藥廠、醫藥公司、國家藥品管理機關等單位。

藥學專業學生畢業后可從事一切與藥物有關的工作：

科研人員在研究所、藥廠的研究部門，從事藥物的研發工作;

醫院藥劑師在醫院藥劑科，從事制劑、質檢、臨床藥學等工作;

藥檢人員在藥檢所從事藥物的質量鑒定和制定相應的質量標準;

公司職員在醫藥貿易公司或制藥企業從事藥品生產、流通及國內外貿易公司、國家藥品管理機關

二、藥學專業的現狀及前景 #from 大學生職業生涯規劃書醫藥學專業來自 end#

據了解，藥學畢業生在選擇工作時主要考慮單位的發展方向和知名度、是否能給自己提供充足的發展空間、工作地點和薪酬水平。目前大學畢業生就業大環境普遍不好，他們的就業也日趨理性和務實。大學生職業生涯規劃書醫藥學專業大學生職業生涯規劃書醫藥學專業。中國藥科大學學生工作處余永久處長說，學校結合市場需求，開設的專業和招生數量與人才需求基本吻合。學生更注重對自己動手能力的培養，能結合自己的專長，選擇適合的工作。

目前藥學類專業專科畢業生期望的月薪為15～XX元，本科生在3元左右，基本符合用人單位愿意支付的薪酬水平，但相比往年有所下降。就業選擇的結構性矛盾突出表現在地域差別上。藥學類畢業生主要選擇在京、津、滬和浙、蘇、粵、魯的沿海城市、省會城市就業，而一些著名的大型藥企由于地域問題，很難招到滿意的人才。大學生職業生涯規劃書醫藥學專業文章大學生職業生涯規劃書醫藥學專業出自wk-78500000544146.html，此鏈接！。

三、藥學專業環境分析(職業生涯規劃swot分析法)

1.優勢(strength)

我國醫院臨床藥學工作自8年開民以來，得到衛生部的重視和支持，并將其工作作為評定醫院等級的一項重要內容，因此在各地大醫院中工作開展得較好，在崗位方面，畢業生到制藥企業從事生產和銷售居多，這方面人才也是企業招聘的主體。現在學醫藥方面的前景很好，目前隨著人們的生活水平在不斷的提高，會對醫療，保健方面的需求會大大增加.所以只要你的技術夠好，前景很好!

2.劣勢(weakness)

由于我國具體的國情和兩階段培養的藥師，在醫院中大多數仍在從事于調劑工作和藥品采購供應以及從事于制劑生產，他們缺乏堅實的臨床醫學和相關臨床知識，難以勝任臨床藥學工作，這與發達國家培養的藥師，在專業相關臨床知識方面差距基大，當前由于經濟利益的驅動，許多醫院領導只關心醫院的經濟利益，因此，難以支持將藥劑科人力、財力投向臨床藥學，故影響了臨床藥學工作的廣泛深入發展，但是隨著醫療衛生體制改革的進行，藥劑科應主動轉變觀念，積極推進開展臨床藥學，在一些有條件的醫院積極推進藥學監護。藥學監護的對象是所在患者，其工作目標和達到的結果是為所有接受藥物治療的患者改善生命質量和身心健康，保證其用藥的安全和有效承擔責任，而臨床藥學的工作對象，大多是住院病人和為臨床醫師提供各種監測數據和資料信息，這些意見采綱與否取決于醫院和患者，此外，臨床藥學的工作目標大多是某些種類的藥物和疾病狀態，所以其工作范圍有一定局限性。因此實施藥學監護，必然要求藥師應具有廣泛的知識能力，才能做好此項工作。

3.機遇(opportunity)

在全國就業形勢不容樂觀的情況下，藥科類畢業生的就業前景仍然普遍看好。醫藥英才網張美玲總經理介紹說，總體來看，藥科類畢業生供小于求，各醫藥公司、制藥廠是吸收這類畢業生的大戶，制藥業對人才的需求是穩中有升。據中國藥科大學、沈陽藥科大學、四川大學華西藥學院、北京大學醫學部藥學院就業工作負責人介紹，近幾年，這幾所學校的畢業生就業率接近1%，總體供需比達到1∶3～1∶4。大學生職業生涯規劃書醫藥學專業職業規劃。沈陽藥科大學學生工作處處長、就業辦公室主任劉彥介紹說，該校的市場營銷本科畢業生的供需比為1∶7，而藥物制劑、天然藥物化學等專業的研究生供需比甚至達到1∶1。目前從教學資源情況看，各學校都沒有擴招計劃。對藥學畢業生來說是一個機會。

第四篇：醫藥學個人簡歷

個人簡歷

姓名：民族：聯系電話：畢業學校：專業：住址：電子信箱：

研究生導師：

執業醫師資格證書：

教育及實習經歷：

培訓經歷：

性別：出生年月：學歷：1

獲獎情況：

2010-2011

2004-2009

200

5擔任職務：

特長及興趣愛好：

在校期間，我參加了基礎部才藝擂臺公益廣告大賽，獲得二等獎； 策劃并參加了“一二·九勿忘國恥”活動；策劃并參加了“考研宣講團”活動，2011年“優秀團支部風采大賽”，獲得一等獎。通過參加各種活動，我懂得了團隊精神的重要性，也提高了我的組織協調能力。多年的校園生活養成了我樂觀向上，大方開朗，熱情務實，善于交流，待人誠懇，工作認真，吃苦耐勞的精神。我愛好閱讀、運動，也喜歡旅游。這些愛好讓我在忙碌緊張的學習之余調劑身心，勞逸結合，提高效率。

自我評價：

我出身于“中醫世家”，在家人的熏陶下立志做一名醫生，獻身于醫學事業！在校期間我學習刻苦，成績優秀，掌握了扎實的醫學基礎理論知識和臨床技能。一年的臨床實習經歷使我提高了分析問題、解決問題的能力。特別是獨立工作、獨立值夜班給我提供了許多實踐機會，增加了我的臨床經驗，使我能更快速準確的對臨床常見病做出診斷和治療。我熱愛“長春中醫藥大學”，是他孕育我成長，所以我希望成為長春中醫藥大學的一名醫生，為中醫藥大學的發展做出貢獻，為醫學事業奮斗終身。

第五篇：醫藥學名詞

二、名詞術語

醫學及藥學名詞應使用全國自然科學名詞審定委員會公布的規范名詞（科學出版社出版）為準。

1.現將常易出錯的不規范名詞糾正如下（括號中為不規范名詞）。

氨基酸轉移酶（轉氨酶）細胞（白血球）紅細胞（紅血球）

作用機制（機理）側支循環（側枝循環）綜合征（綜合癥）

膽固醇（膽甾醇）單核－吞噬細胞系統（網狀內系統）

低鉀血癥（低血鉀癥）高脂血癥（高血脂癥）內鏡（內窺鏡）

固醇（甾醇）發紺（紫紺）反胃（返胃）

分枝桿菌（分支桿菌）肺源性（肺原性）肺梗死（肺梗塞）

腦出血（腦溢血）腦梗死（腦梗塞）心肌梗死（心肌梗塞）

腦卒中（中風）放射性核素（同位素）功能（機能）

肝硬化（肝硬變）核糖體（核蛋白體）晶狀體（晶體）

膠原纖維（膠元纖維）假膜（偽膜）咳痰（咯痰）

咯血（咳血）抗生素（抗菌素）磷脂酰膽堿（卵磷脂）

黏膜（粘膜）清蛋白（白蛋白）期前收縮（早博）

妊娠高血壓綜合征（妊娠中毒癥）三酰甘油（甘油三脂）

腎衰竭（腎功能衰竭）食欲缺乏（食欲不振）食管（食道）

嗜酸（堿）性細胞（嗜酸（堿）性白細胞）同工酶（同功酶）

糖原（糖元）畏食（厭食）糖皮質激素（糖皮質類固醇）

圍生期（圍產期）下丘腦（丘腦下部）心排出量（心輸出量）

心源性（心原性）血紅蛋白（血色素）血流動力學（血液動力學）

藥源性（藥原性）醫源性（醫原性）真菌（霉菌）

原發性高血壓（高血壓）